A static progressive splint with a wrist hinge hand portion was fabricated over the electromesh glove. The electromesh glove was worn for an additional 30 minutes. An electromesh glove was fitted with repositioning of proximal electrodes utilizing channel 2, after stimulation of the antagonistic musculature permitted positioning of the wrist in neutral. The negative electrode was placed over motor point with cycle rates used to produce tetany at 25–50 pps (the minimum rate that produced a good tetanized contraction dependent on tone) (Kahn 1987). As channel intensity is decreasing, channel 2 started increasing intensity, via the same pattern. Channel 1 gradually increased intensity for 0.5 seconds, and then held a set intensity for 5.0 seconds and then decreased intensity over 0.5 seconds. Channel 2 started after channel 1 had completed the cycle. This configuration was applied for 30 minutes using an alternating ramped burst program with an asymmetrical waveform for same musculature. Channel 2 was applied to the wrist extensors to facilitate wrist position and digital extension for appropriate pre-contact grasp formation. Channel 1 was applied to the triceps to facilitate elbow extension for increased reach envelope for improved functional use of the hand and ease for splinting. NMES was used to stimulate the antagonistic muscle groups consisting of extensor carpi radialis longus and brevis (ECRL/B), extensor digitorum communis (EDC), and triceps. As a result, working with a physical therapist to identify the optimal solution is your best bet.Īs you continue to exercise, you should ultimately be able to attain normal mobility and function - or as near to it as feasible.Method: Three patients presented after left-sided flexor synergistic tone at approximately 60 degrees of wrist flexion with digital composite flexion, elbow at 40 degrees of flexion without volitional control. Of course, this may be difficult, particularly when motions are abnormal. The more you exercise your injured muscles, the more your brain can generate new neural connections that will allow muscle groups to communicate again. When severe spasticity and synergistic movement prohibit a stroke survivor from moving at all, it’s evident that any sort of movement is preferable to none when you’re giving it your all and concentrating on the correct form every time.Īs long as you do your hardest to employ proper form every time you exercise, you will continue to promote neuroplasticity and improve. Most therapists would agree that doing rehab activities poorly (i.e., with synergistic movement patterns) is not desirable since it may perpetuate these bad movement patterns. It is possible to have several kinds of synergy patterns, such as flexion synergy patterns (which were explained before), extension synergy patterns, and other kinds of synergy patterns, depending on the particular muscle groups that are cooperating with one another. These patterns can be caused by the brain trying to adapt to its new state. However, in some instances, such as after a stroke or another neurological condition, the brain’s ability to control individual muscle groups may be impaired, which can lead to the development of abnormal or compensatory synergy patterns. This is because the body naturally employs particular muscle groups to produce certain motions in cooperation with other groups of muscles. Synergy patterns are not exclusive to people who have had a stroke they may also be seen in healthy persons. To put it another way, when a synergy pattern is present, several muscles collaborate in order to carry out a certain action, as opposed to each muscle acting separately to carry out the movement. The term “synergy patterns” refers to the manner in which several muscle groups in the body create movement by cooperating with one another, also known as “synergizing.” They are distinguished from the isolated activity of individual muscle groups by the coordinated action of many muscle groups rather than by the activity of individual muscle groups acting alone.
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